Extracardiac coronary steal induced by upper limb hyperemia: a feature of internal mammary artery arteriogenesis

Abstract Introduction Function of naturally existing internal mammary artery (IMA)-to-coronary anastomoses has been shown by augmented blood supply to the coronary collateral circulation in response IMA occlusion. Theoretically, this functional connection is invertible and can cause steal, verification whose hypothesis would provide alternate proof mentioned evidence. Method This was a prospective observational study including 40 patients with chronic syndrome occlusion, propensity score matched controls (placebo group) without Primary endpoint inter-group difference function (i.e., flow index, CFI) as obtained at 30, 45 60 seconds following proximal balloon CFI ratio between simultaneous mean occlusive pressure divided aortic both subtracted central venous pressure. To provoke steal phenomenon, upper limb hyperemia induced arm cuff deflation 5-minute supra-systolic inflation ipsilateral sensor-wired release immediately after first measurement. Results Between second CFI-measurement, change CFI@45s minus CFI@30s) absent verum group while there recruitment placebo group: 0.000±0.023 +0.009±0.013, respectively; p=0.032. Conclusion Among artificial induction provokes extracardiac expressed temporarily it normally takes place hyperemia. Funding Acknowledgement Type funding sources: Public Institution(s). Main source(s): Swiss National Sciences Foundation